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Additional analyses showed that testosterone reduces levels of certain transcription factors (regulatory proteins) that ordinarily prevent Module 52 genes from “turning on.” In other words, higher testosterone levels result in more Module 52 expression. Several Module 52 genes have known immune-system connections; activation of one of these genes, for example, results in the accelerated differentiation of cells whose job it is to suppress, rather than foster, immune response. These connections make the interactions of the genes with testosterone an intriguing target of further exploration by immunologists, physiologists and drug researchers, Davis said.

Hi, I have mild cardiovascular disease but am only in my mid-fifties. I am very slim, extremely fit, vegan, with a great HDL and triglycerides profile and fabulous HDL/LDL ratio (so clearly my problem is not dietary) but have high LDL cholesterols for familial/genetic reasons as well as high BP. Doctors have tried me with statins (just 8 weeks’ use caused muscle damage and inflammation as well as abnormal liver function tests) and I cannot take ezetimibe (ezetrol) to lower my LDLs as it causes tingling in my extremities and severe depression (suicidal ideation.) I have some pueraria mirifica, bought originally for breast enhancement and daren’t use it, in case it makes my atherosclerotic plaques worse, because my GP said I can’t take HRT owing to my existing cardiovascular plaques. I’ve heard that PM improves your lipids profile, though (and in my case could resolve some of the breast and skin looseness problems I’m having post-menopause) and wondered if it might be safe to take in patients with existing atherosclerosis whereas conventional HRT is not. Does anyone in a similar position or know anything about this?

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